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" "(4) With that trade-off as a fundamental constraint, selection adjusts telomere lengths—longer telomeres increasing the capacity for repair, shorter telomeres increasing tumor resistance.
(born 21 February 1969) is an American evolutionary biologist and podcaster who came to national attention during the 2017 Evergreen State College protests. He is among the people referred to collectively as the "," a term coined by Weinstein's brother .
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I... struggle to find language that is strong enough for the horror of vaccinating children in this case, because children suffer a greater risk of long-term effects... this is earlier in their development, therefore it impacts systems that are still forming. They tolerate COVID well, and so the benefit to them is very small, and so the only argument for doing this is that they may cryptically be carrying more COVID than we think, and therefore they may be integral to the way virus spreads to the population. But if that's the reason that we're inoculating children... we were doing it to protect old, infirm people who are the most likely to succumb... What society puts children in danger, robs children of life, to save old infirm people. That's upside-down.
[A]ll of our environmental problems look like... somebody making a profit for degrading what belongs to the rest of us. ...[T]his behavior should not be allowed within a marketplace. We are fracturing the world. We are liquidating it, we are draining it, we are denuding it, we are over-exploiting it. It is apparent to anyone... The idea that this should not be allowed is transparent.
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[My] work... looked at the fact that shortening was being looked at by two different groups... by people interested in counteracting the aging process, and... in exactly the opposite fashion, by people who were interested in tumorigenesis in cancer. ...Tumors ...always had active, that's the enzyme that lengthens our telomeres. So those folks were interested in bringing about a halt in the lengthening of telomeres... to counteract cancer, and the folks that were studying the process were interested in lengthening telomeres... to generate greater repair capacity. ...[M]y point was evolutionarily speaking this looks like a pleiotropic effect, that the genes which create the tendency of the cells to be limited in their capacity to replace themselves, are providing a benefit in youth... that we are largely free of tumors and cancer at the inevitable late life cost that we grow feeble and inefficient, and eventually die. ...[T]hat matches a very old hypothesis in evolutionary theory by somebody I was fortunate enough to know, George Williams...